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Spinal Deformities Dr. Budak Akman T.C Yeditepe University Orthopaedics and Traumatology.

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... konulu sunumlar: "Spinal Deformities Dr. Budak Akman T.C Yeditepe University Orthopaedics and Traumatology."— Sunum transkripti:

1 Spinal Deformities Dr. Budak Akman T.C Yeditepe University Orthopaedics and Traumatology

2 Physical Examination of Spine

3 Fizik Muayene İnspeksiyon Palpasyon Hareket açıklığı Tendon refleksleri Nörolojik Muayene

4 İnspeksiyon Asimetri - Pelvik Oblisite – Pelvik rotasyon, alt ekstremitede uzunluk farkı Lordoz veya kifoz artışı Skolyoz Café au lait spotları-nodüller Kıllanma – kırmızı şarap lekesi Yürüyüş muayenesi

5 Palpasyon Spinöz proçeslerde basamak Paraspinal Adale spazmı – pozisyon Lokal hassasiyet noktaları Tetik noktaları Sakroiliak eklem muayenesi

6 ROM (=Hareket açıklığı ) Öne fleksiyon : Parmak ucu yer mesafesi ölçümü (N:10cm) Ekstansiyon : Lordoz artışı (değişik derecelerde) (N:20˚-30˚) Lateral fleksiyon : Parmak uçlarının diz eklemine göre pozisyonları (N:20˚-30˚)

7 Nörolojik Muayene Motor (spastisiteye dikkat) Duysal (hafifçe dokunma ve iğne batırma) Refleksler L3, S1, S2-4, Babinski

8 DUYU-MOTOR DERMATOMDUYU TESTİMOTOR TESTREFLEKS TESTİ L1-L2Anteromedial uyluk İliopsoas- L3Patella proksimaliQuadricepsPatella refleksi L3 L4Kruris-ayakbileği mediali Tibialis anterior- L5Krurisin laterali- ayak dorsali Ekstansör Hallucis longus S1Baldır posterioru- topuk GastrosoleusAşil refleksi S1 S2Femur posterioruRektal kontraksiyon S3-S4-S5Perianal bölgeRektal kontraksiyon


10 Özgül Testler Düz bacak kaldırma Çapraz düz bacak kaldırma testi Laseque testi Valsalva Manerası Femoral GermeTesti L5-S1 L3-L4

11 SPONDYLOLYSİS AND SPONDYLOLİSTHESİS Spondylolysis and Spondylolisthesis – Etiology Spondylolysis refers to degeneration of the vertebrae due to congenital weakness (stress fracture results) Slipping of one vertebrae above or below another is referred to as spondylolisthesis and is often associated with a spondylolysis – Signs and Symptoms Spondylolysis begins unilaterally Pain and persistent aching, low back stiffness with increased pain after activity Frequent need to change position Full ROM w/ some hesitation in regards to flexion Localized tenderness and some possible segmental hypermobility Step off deformity may be present

12 Congenital:Congenital dysplasia of facet joınt L5-S1 Isthmic: Defect of Pars interarticularıs (EN SIK) Degenerative: facet arthrosis leading to subluxation Traumatic: Acut Pathologic: Tm, enfection, paget disease… Post surgical: iatrogenic SPONDYLOLYSİS AND SPONDYLOLİSTHESİS CLASSIFICATION

13 Spondylolysis and Spondylolisthesis



16 – Management Bracing and occasionally bed rest for 1-3 days will help to reduce pain Major focus should be on exercises directed as controlling or stabilizing hypermobile segments Progressive trunk strengthening, dynamic core strengthening, concentration on abdominal work Braces can also be helpful during high level activities Increased susceptibility to lumbar strains and sprains and thus vigorous activity may need to be limited

17 Spinal Deformities

18 Spinal deformities occur in either coronal or sagittal plane

19 What is scoliosis? Lateral curvature of the spine >10º accompanied by vertebral rotation Idiopathic scoliosis - Multigene dominant condition with variable phenotypic expression & no clear cause Multiple causes exist for secondary scoliosis

20 Scoliosis classification 1-Structural: Idiopatic Congenital neuromuscular Neurofibromatosis Osteochondrodystrophyos Metabolic 2-Non-structural: (postural, histeric, herniopaty)

21 Idiopathic Scoliosis “Classification” Age at Onset: Infantile: age birth to 3 years Juvenile: age 4 to 10 years Adolescent: age 11 to 17 years Adult: age 18 years up

22 Idiopathic Scoliosis “Etiology” Remains unknown Several studies have attempted to look into this and various factors have been postulated: genetic, tissue deficiencies, vertebral growth abnormalities, and central nervous system theories

23 Idiopathic Scoliosis “Genetic Factors” Risenborough found a 11.1% incidence of scoliosis in first born relatives of patients with idiopathic scoliosis Twins show a concordance of scoliosis with an incidence of 92% monozygotic and 63% dizygotic

24 Secondary causes for scoliosis: Inherited connective tissue disorders - Ehler’s Danlos syndrome - Marfan syndrome - Homocystinuria

25 Secondary causes for scoliosis: Neurologic disorders Tethered cord syndrome Syringomyelia Spinal tumor Neurofibromatosis Muscular dystrophy Cerebral palsy Polio Friedeich’s ataxia Familial dysautonomia Werdnig-Hoffman disease

26 Secondary causes for scoliosis: Musculoskeletal disorders »Leg length discrepancy »Developmental hip dysplasia »Osteogenesis imperfecta »Klippel-Feil syndrome

27 Characteristics of idiopathic scoliosis: Present in 2 - 4% of kids aged 10 – 16 years Ratio of girls to boys with small curves ( 30º the ratio is 10:1 Scoliosis tends to progress more often in girls (so girls with scoliosis are more likely to require treatment) Toracal curve (right) Lomber curve (left)

28 Natural history of scoliosis Of adolescents diagnosed with scoliosis, only 10% have curve progression requiring medical intervention Three main determinants of curve progression are: (1) Patient gender (2) Future growth potential (3) Curve magnitude at time of diagnosis

29 Natural history of scoliosis Assessing future growth potential using Tanner staging: Tanner stages 2-3 (just after onset of pubertal growth) are the stages of maximal scoliosis progression

30 Natural history of scoliosis Assessing growth potential using Risser grading: - Measures progress of bony fusion of iliac apophysis - Ranges from zero (no ossification) to 5 (complete bony fusion of the apophysis) - The lower the grade, the higher the potential for progression

31 Risser 1 = 25% Capping. Risser 2 = 50% Capping. Risser 3 = 75% Capping. Risser 4 = 100% Capping. Risser 5 = 100% Capping + Fusion. Risser 1 = 25% Capping. Risser 2 = 50% Capping. Risser 3 = 75% Capping. Risser 4 = 100% Capping. Risser 5 = 100% Capping + Fusion. Line Of Risser Risser 2


33 Natural history of scoliosis Back pain not significantly higher in pts with scoliosis Curves in untreated adolescents with curves < 30 º at time of bony maturity are unlikely to progress Curves >50 º at maturity progress 1º per year Up to 19% of females with curves >40 º have significant psychological illness Life-threatening effects on pulmonary function do not occur until curve is >100 º (ie: Cor pulmonale)

34 Adam’s forward bend test For this test, the patient is asked to lean forward with his or her feet together and bend 90 degrees at the waist. The examiner can then easily view from this angle any asymmetry of the trunk or any abnormal spinal curvatures.

35 Screening hints: Shoulders are different heights – one shoulder blade is more prominent than the other Head is not centered directly above the pelvis Appearance of a raised, prominent hip Rib cages are at different heights Uneven waist Changes in look or texture of skin overlying the spine (dimples, hairy patches, color changes) Leaning of entire body to one side Cavus feet

36 Red flags on PE: Left-sided thoracic curvature Pain Significant stiffness Abnormal neurologic findings Stigmata of other clinical syndromes associated with curvature Juvenil scoliosis MRI

37 Measure spinal curvature using Cobb method : -Choose the most tilted verterbrae above & below apex of the curve. - Angle b/t intersecting lines drawn perpendicular to the top of the superior vertebrae and bottom of the inferior vertebrae is the Cobb angle.

38 Measure spinal curvature using Cobb method:


40 Treatment Decisions Accepted Standards


42 Brace Treatment for Scoliosis Most common is Boston brace Braces have 74% success rate at halting curve progression (while worn) Bracing does not correct scoliosis, but may prevent serious progression Usually worn until patient reaches Risser grade 4 or 5

43 Brace Treatment for Scoliosis Of patients with 20 º - 29 º curves, only 40% of those wearing braces ultimately required surgery, compared to 68% of those not wearing back braces Length of wearing time correlates with outcome (At least 16 hrs per day leads to best chance of preventing curve progression)

44 Idiopathic Scoliosis SURGICAL CORRECTION GOALS  Reduce the magnitude of the curve  Obtain fusion to prevent progression  Create a well-balanced spine

45 Idiopathic Scoliosis SURGICAL CORRECTION INDICATIONS Curves over 45 degrees Trunk deformity(rotation) Trunk balance Progressive curves despite bracing Congenital scoliosis Neurologic symptoms

46 Idiopathic Scoliosis GENERAL GUIDELINES FOR TREATMENT OF SCOLIOSIS Under 20 degree’s: observe 20 to 30 degree’s: observe with frequent follow-up; progression then brace 30 to 45 degree’s: brace unless Risser 4/5 then observe 45 plus degree’s: instrumentation

47 Scoliosis treatment plan CurveRisserTherapy 0-25Immatureobserve 25-30immaturebrace 30-45immaturebrace >45immaturesurgery >50maturesurgery

48 Surgical Treatment for Scoliosis Curves in growing children greater than 40 º require a spinal fusion (Risser grade 0 to 1 in girls and Risser 2 or 3 in boys) Skeletally mature patients can be observed until their curves reach 50 º Posterior spinal fusion is best choice for thoracic curves Anterior spinal fusion is best treatment for thoracolumbar and lumbar curves

49 Surgical Treatment for Scoliosis

50 Scoliosis Adolescent idiopathic scoliosis Structural scoliosis presenting at or about the onset of puberty and before maturity 80 % of cases of idiopathic scoliosis Mostly (90%) in girls Predictors of progression very young age marked curvature Risser sign


52 Congenital Scoliosis Congenital scoliosis Due to congenital anomalous vertebral development Hemivertebrae Wedged vertebrae Fused vertebrae Absent or fused ribs Treatment Early fusion in progressive curves

53 Congenital Scoliosis

54 Neuromuscular Scoliosis Neuromuscular scoliosis Causes Poliomyelitis Cerebral palsy Syringomyelia Friedrich’s ataxia Muscular dystrophies Typical paralytic curve is long, convex towards the side with weaker muscles

55 Kyphosis Postural Congenital Scheuermann’s disease T1-T12: 20 - 40°

56 Kyphosis Postural POSTURAL YAPISAL

57 Kyphosis Congenital

58 Kyphosis Scheuermann’s disease Cobb angle >45° with wedging of 5 or more of at least 3 adjacent apical vertebrae vertebral end plate irregularities Etiology :Unknown Incidence:1% of general population with slight female dominance






64 Spinal stenoz Spinal kanalın daralma yolu ile nöral doku basılarına neden olan klinik durumdur. Kanal çapında merkezi daralmalar olabileceği gibi lateral reseslerde de kök basılarına yol açan sıkışmalar gözlenebilir.


66 Spinal Stenoz ( Klinik yaklaşım ) Etyoloji: – Konjenital daralma – Spondilolistezis – Travma – Yaşlanmaya bağlı dejeneratif değişiklikler – Diğer Semptomlar

67 Etyolojik Yaklaşım Konjenital / Edinsel kanal darlığı İdiopatik: pedikül gelişiminde duraklama Spondilolistezis, Paget hastalığı, Akondroplazi gibi kemik hastalıklarına bağlı olarak gelişebilirler.

68 Ağrı: Ağrı bölgesi (bel,bacak) Şikayetlerin tanımlanması: Uyuşukluk,ağrı,güçsüzlük Ağrının tipi – Ayak ağrısı, saplanır tarzda ağrı, elektriklenme – Devamlı, fasılalı Devamlılık Akut, subakut kronik Başlangıç, ani, yavaş, travmatik

69 Ağrı Dışı Semptomatoloji Nörojenik klaudikasyo Radikülopati İkisinin birlikteliği

70 Klinik Değerlendirme Nörojenik klaudikasyo semptomatik stenozlu olgularda %50-62 oranında ortaya çıkar. Nörojenik klaudikasyo klinikte karşımıza bel kalça uyluk ve baldır ağrısı olarak karşımıza çıkar.

71 Risk Faktörleri: Obezite (3 x risk artışı) Sigara (öksürme ile ) Mesleki zorlanma – Tekrarlayıcı hareket, vibrasyon veya burulma hareketi – Uzun süreli araç kullanımı – Sedanter yaşam

72 Spinal stenozun ayırıcı tanısı Dejeneratif disk hastalığı Faset artropatisi Spondilolistezis Spondilolizis Herniye disk Diskojenik ağrı

73 Radiküler Semptomlar Ağrının dağılımı, taraf bulgusu ve niceliği Dermatomal ağrı dağılımı veya uyuşukluk ve sızlama – L3 or L4 - anterior uyluk ağrısı – L5 -ayak sırtı ve başparmak – S1 -posterior baldır, topuk, ayak dış yanı Bacak Ağrısı > Bel Ağrısı!

74 Radiküler Semptomlar Artmış ağrı cevabı: – Fleksiyon – Oturma – Öksürme,hapşırma,ıkınma Ağrıda azalma: – Supin pozisyonda yatma – Dizlerde fleksiyona getirme – Ayakta durma

75 Tedavi Konservatif tedavi – FTR – Korse – NSAI – Kilo verme Cerrahi tedavi – Dekompresyon – Füzyon


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